goscripttm reverse transcriptase a5003 (Promega)
90
Structured Review
Promega
goscripttm reverse transcriptase a5003
Goscripttm Reverse Transcriptase A5003, supplied by Promega, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/goscripttm+reverse+transcriptase/goscripttm+reverse+transcription+system/pmc10214193__pnas__2217826120__sapp-118-3-21
Average 90 stars, based on 1 article reviews
Goscripttm Reverse Transcriptase A5003, supplied by Promega, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/goscripttm+reverse+transcriptase/goscripttm+reverse+transcription+system/pmc10214193__pnas__2217826120__sapp-118-3-21
Average 90 stars, based on 1 article reviews
goscripttm reverse transcriptase a5003 - by Bioz Stars,
2026-10
90/100 stars
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Reverse Transcription:Article Title: Generation of bovine iPSCs from fetal fibroblasts for in vitro myogenesis and cultured meat Article Snippet: The Ribonucleic Acid (RNA) samples were analyzed regarding quantity and quality using a spectrophotometer (NanoDrop Lite Plus Spectrophotometer, cat. NDLPLUSULCA). .. Reverse transcription of the extracted RNA was performed using the commercial Article Title: Comparative study of CYP450 gene regulation in human pluripotent stem cell-derived liver 2D cell and 3D organoid models. Article Snippet: Human pluripotent stem cell (hPSC)-derived hepatic models, including 2D hepatocyte-like cells (2D HLCs) and hepatic organoids (HOs), are valuable in vitro models for evaluating the safety and efficacy of drugs.. However, 2D HLCs show limited expression and activity of drug-metabolizing enzymes, particularly cytochrome P450 (CYP450), which are involved in detoxification, a major liver function.. HOs have more mature properties than 2D HLCs, particularly enhanced CYP450 gene expression. Article Title: Synthetic homoisoflavane derivatives suppress the growth of colorectal cancer cells by disturbing microtubule dynamics. Article Snippet: Microtubules, composed of tubulin proteins, have a crucial role in various cellular processes, including cell cycle regulation.. The dynamic instability of microtubules has been an attractive target for cancer therapy.. This study investigated two potent anticancer drug candidates, homoisoflavane derivatives SH-19021 and SHA-035, and evaluated their potential as microtubule-targeting agents (MTAs) in human colorectal cancer cells. Article Title: Isoform-specific function of NSD3 in DNA replication stress confers resistance to PARP inhibitors in prostate cancer. Article Snippet: Article Isoform-specific function of NSD3 in DNA replication stress confers resistance to PARP inhibitors in prostate cancer Graphical abstract Highlights • NSD3S, but not NSD3L, contributes to the resistance of prostate cancer to PARPi • The CUL3ZBTB2 E3 ubiquitin ligase complex specifically targets NSD3S for degradation • NSD3S protects against MRE11-mediated degradation of the stalled replication fork • NSD3 PROTAC enhances the sensitivity of prostate cancer to PARPi Authors Shouhai Zhu, Huanyao Gao, Dan Jiang, ..., Xinyi Tu, Liewei Wang, Zhenkun Lou Correspondence tu.xinyi@mayo.edu (X.T.), wang.liewei@mayo.edu (L.W.), lou.zhenkun@mayo.edu (Z.L.). In brief In this study, Zhu et al. demonstrate that upregulated NSD3S confers acquired PARPi resistance in prostate cancer cells.. ATR drives the localization of NSD3S to stalled replication forks, where it protects nascent DNA from extensive degradation and ensures fork stabilization by antagonizing PTIP-dependent recruitment of the MRE11 nuclease. Article Title: Mealworm hydrolysate ameliorates dexamethasone-induced muscle atrophy via sirtuin 1-mediated signaling and Akt pathway Article Snippet: RNA was extracted from the muscle tissues and C2C12 cells using a total RNA extraction kit (iNtRON Biotechnology, Gyeonggi-do, Korea) according to the manufacturer’s instructions. .. Complementary DNA synthesis was performed using Article Title: The renoprotective effects of tadalafil on ischemia-reperfusion injury during partial nephrectomy in an animal model. Article Snippet: RNA was extracted using a Maxwell® RSC simply RNA Cells Kit and a MaxwellTM instrument (Promega, Madison, WI, USA). .. After RNA quantification, RNA (1 ug) was reversed transcribed into cDNA using Article Title: Polyester artificial ligament modified with a polyphenol-zinc layer for controlled and targeted release of ciprofloxacin Article Snippet: .. Isolated RNA was reverse transcribed into complementary DNA (cDNA) using Isolation:Article Title: Synthetic homoisoflavane derivatives suppress the growth of colorectal cancer cells by disturbing microtubule dynamics. Article Snippet: Microtubules, composed of tubulin proteins, have a crucial role in various cellular processes, including cell cycle regulation.. The dynamic instability of microtubules has been an attractive target for cancer therapy.. This study investigated two potent anticancer drug candidates, homoisoflavane derivatives SH-19021 and SHA-035, and evaluated their potential as microtubule-targeting agents (MTAs) in human colorectal cancer cells. Article Title: Polyester artificial ligament modified with a polyphenol-zinc layer for controlled and targeted release of ciprofloxacin Article Snippet: .. Isolated RNA was reverse transcribed into complementary DNA (cDNA) using Expressing:Article Title: Synthetic homoisoflavane derivatives suppress the growth of colorectal cancer cells by disturbing microtubule dynamics. Article Snippet: Microtubules, composed of tubulin proteins, have a crucial role in various cellular processes, including cell cycle regulation.. The dynamic instability of microtubules has been an attractive target for cancer therapy.. This study investigated two potent anticancer drug candidates, homoisoflavane derivatives SH-19021 and SHA-035, and evaluated their potential as microtubule-targeting agents (MTAs) in human colorectal cancer cells. Real-time Polymerase Chain Reaction:Article Title: Synthetic homoisoflavane derivatives suppress the growth of colorectal cancer cells by disturbing microtubule dynamics. Article Snippet: Microtubules, composed of tubulin proteins, have a crucial role in various cellular processes, including cell cycle regulation.. The dynamic instability of microtubules has been an attractive target for cancer therapy.. This study investigated two potent anticancer drug candidates, homoisoflavane derivatives SH-19021 and SHA-035, and evaluated their potential as microtubule-targeting agents (MTAs) in human colorectal cancer cells. other:Article Title: Oxidative DNA Damage and Repair Dynamics in Multiple Sclerosis: Insights from Comet Assay Kinetics, Base Excision Repair Gene Expression, and Genotype Analysis. Article Snippet: Subsequently, according to the manufacturer’s instructions, 100 ng of Proliferation Assay:Article Title: Isoform-specific function of NSD3 in DNA replication stress confers resistance to PARP inhibitors in prostate cancer. Article Snippet: Article Isoform-specific function of NSD3 in DNA replication stress confers resistance to PARP inhibitors in prostate cancer Graphical abstract Highlights • NSD3S, but not NSD3L, contributes to the resistance of prostate cancer to PARPi • The CUL3ZBTB2 E3 ubiquitin ligase complex specifically targets NSD3S for degradation • NSD3S protects against MRE11-mediated degradation of the stalled replication fork • NSD3 PROTAC enhances the sensitivity of prostate cancer to PARPi Authors Shouhai Zhu, Huanyao Gao, Dan Jiang, ..., Xinyi Tu, Liewei Wang, Zhenkun Lou Correspondence tu.xinyi@mayo.edu (X.T.), wang.liewei@mayo.edu (L.W.), lou.zhenkun@mayo.edu (Z.L.). In brief In this study, Zhu et al. demonstrate that upregulated NSD3S confers acquired PARPi resistance in prostate cancer cells.. ATR drives the localization of NSD3S to stalled replication forks, where it protects nascent DNA from extensive degradation and ensures fork stabilization by antagonizing PTIP-dependent recruitment of the MRE11 nuclease. In Situ:Article Title: Isoform-specific function of NSD3 in DNA replication stress confers resistance to PARP inhibitors in prostate cancer. Article Snippet: Article Isoform-specific function of NSD3 in DNA replication stress confers resistance to PARP inhibitors in prostate cancer Graphical abstract Highlights • NSD3S, but not NSD3L, contributes to the resistance of prostate cancer to PARPi • The CUL3ZBTB2 E3 ubiquitin ligase complex specifically targets NSD3S for degradation • NSD3S protects against MRE11-mediated degradation of the stalled replication fork • NSD3 PROTAC enhances the sensitivity of prostate cancer to PARPi Authors Shouhai Zhu, Huanyao Gao, Dan Jiang, ..., Xinyi Tu, Liewei Wang, Zhenkun Lou Correspondence tu.xinyi@mayo.edu (X.T.), wang.liewei@mayo.edu (L.W.), lou.zhenkun@mayo.edu (Z.L.). In brief In this study, Zhu et al. demonstrate that upregulated NSD3S confers acquired PARPi resistance in prostate cancer cells.. ATR drives the localization of NSD3S to stalled replication forks, where it protects nascent DNA from extensive degradation and ensures fork stabilization by antagonizing PTIP-dependent recruitment of the MRE11 nuclease. Proximity Ligation Assay:Article Title: Isoform-specific function of NSD3 in DNA replication stress confers resistance to PARP inhibitors in prostate cancer. Article Snippet: Article Isoform-specific function of NSD3 in DNA replication stress confers resistance to PARP inhibitors in prostate cancer Graphical abstract Highlights • NSD3S, but not NSD3L, contributes to the resistance of prostate cancer to PARPi • The CUL3ZBTB2 E3 ubiquitin ligase complex specifically targets NSD3S for degradation • NSD3S protects against MRE11-mediated degradation of the stalled replication fork • NSD3 PROTAC enhances the sensitivity of prostate cancer to PARPi Authors Shouhai Zhu, Huanyao Gao, Dan Jiang, ..., Xinyi Tu, Liewei Wang, Zhenkun Lou Correspondence tu.xinyi@mayo.edu (X.T.), wang.liewei@mayo.edu (L.W.), lou.zhenkun@mayo.edu (Z.L.). In brief In this study, Zhu et al. demonstrate that upregulated NSD3S confers acquired PARPi resistance in prostate cancer cells.. ATR drives the localization of NSD3S to stalled replication forks, where it protects nascent DNA from extensive degradation and ensures fork stabilization by antagonizing PTIP-dependent recruitment of the MRE11 nuclease. Mutagenesis:Article Title: Isoform-specific function of NSD3 in DNA replication stress confers resistance to PARP inhibitors in prostate cancer. Article Snippet: Article Isoform-specific function of NSD3 in DNA replication stress confers resistance to PARP inhibitors in prostate cancer Graphical abstract Highlights • NSD3S, but not NSD3L, contributes to the resistance of prostate cancer to PARPi • The CUL3ZBTB2 E3 ubiquitin ligase complex specifically targets NSD3S for degradation • NSD3S protects against MRE11-mediated degradation of the stalled replication fork • NSD3 PROTAC enhances the sensitivity of prostate cancer to PARPi Authors Shouhai Zhu, Huanyao Gao, Dan Jiang, ..., Xinyi Tu, Liewei Wang, Zhenkun Lou Correspondence tu.xinyi@mayo.edu (X.T.), wang.liewei@mayo.edu (L.W.), lou.zhenkun@mayo.edu (Z.L.). In brief In this study, Zhu et al. demonstrate that upregulated NSD3S confers acquired PARPi resistance in prostate cancer cells.. ATR drives the localization of NSD3S to stalled replication forks, where it protects nascent DNA from extensive degradation and ensures fork stabilization by antagonizing PTIP-dependent recruitment of the MRE11 nuclease. Mass Spectrometry:Article Title: Isoform-specific function of NSD3 in DNA replication stress confers resistance to PARP inhibitors in prostate cancer. Article Snippet: Article Isoform-specific function of NSD3 in DNA replication stress confers resistance to PARP inhibitors in prostate cancer Graphical abstract Highlights • NSD3S, but not NSD3L, contributes to the resistance of prostate cancer to PARPi • The CUL3ZBTB2 E3 ubiquitin ligase complex specifically targets NSD3S for degradation • NSD3S protects against MRE11-mediated degradation of the stalled replication fork • NSD3 PROTAC enhances the sensitivity of prostate cancer to PARPi Authors Shouhai Zhu, Huanyao Gao, Dan Jiang, ..., Xinyi Tu, Liewei Wang, Zhenkun Lou Correspondence tu.xinyi@mayo.edu (X.T.), wang.liewei@mayo.edu (L.W.), lou.zhenkun@mayo.edu (Z.L.). In brief In this study, Zhu et al. demonstrate that upregulated NSD3S confers acquired PARPi resistance in prostate cancer cells.. ATR drives the localization of NSD3S to stalled replication forks, where it protects nascent DNA from extensive degradation and ensures fork stabilization by antagonizing PTIP-dependent recruitment of the MRE11 nuclease. DNA Synthesis:Article Title: Mealworm hydrolysate ameliorates dexamethasone-induced muscle atrophy via sirtuin 1-mediated signaling and Akt pathway Article Snippet: RNA was extracted from the muscle tissues and C2C12 cells using a total RNA extraction kit (iNtRON Biotechnology, Gyeonggi-do, Korea) according to the manufacturer’s instructions. .. Complementary DNA synthesis was performed using |